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Concurrently, downregulation of STAMBP and NEP may impair protein clearance mechanisms, exacerbating proteotoxic stress and -amyloid (A) deposition [52], ultimately triggering neuronal apoptosis, which aligns with our previous observation of reduced MAP2-positive neurons

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Any references to tissue modeling, collagen, or aging-related pathways are provided only for research context and do not imply cosmetic or clinical use

The most common research areas linked to Epithalon include longevity studies, cellular aging research, telomere research, sleep-related aging pathways, immune function studies, and overall cellular health investigations

Studies also report reduced inflammatory cytokine activity, improved microvascular integrity, and beneficial effects on pain modulation through peripheral and dopaminergic mechanisms